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Paper published by Miss Hengyan Zhang:
Original Research
![Noteworthy comment: Knockdown and overexpression of Ufm1 in cell line and animal model in the study elucidate a new biological function of Ufm1 that is increased in diabetic mice and mediates inflammatory responses through modulating the ubiquitination and degradation of IκBα, suggesting that down-regulation of Ufm1 is an effective strategy to prevent development of type 2 diabetes and its complications.](assets/img/article_icons/noteworthy.png)
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Ubiquitin-Fold Modifier-1 Participates in the Diabetic Inflammatory Response by Regulating NF-κB p65 Nuclear Translocation and the Ubiquitination and Degradation of IκBα
Hu X, Zhang H, Zhuang L, Jin G, Yang Q, Li M, Sun W, Chen F
Drug Design, Development and Therapy 2020, 14:795-810
Published Date: 25 February 2020